A structural disease wherein the vessels supplying blood to the brain are inadequately “decoupled”. This can result in increased bleeding, stroke and brain damage. In some cases it may result in dementia, learning and memory deficits.
There are two types of human uveitis, DPU and cPP. In DPU, the inflammation starts within the lining of the vessel. In cPP, it seems to originate outside of the wall of the vessel. This may include damage to the plaques or deposits that form in the vessel’s walls.
There are three types of inflammation in human uveitis: intracellular, proliferative, and inflammatory.
In lesions caused by intracellular inflammation, the surface of the vessel becomes inflamed as a result of damage to the inner lining of the vessel or inflammatory cell activity. The inflammation can infiltrate the veins of the arm and then may spread to the eye causing infection.
Depending on the severity of the patient’s disease and other, undiagnosed medical conditions, ulcers may spontaneously heal. If this happens, it may indicate that the injury has been repaired within the vessel walls. However, if the ulcer persists, the vessel walls can be damaged or damaged.
In derangements of the lining, it appears that further damage to the vessel walls has occurred.
In derangements of the lining, it appears that further damage to the vessel walls has occurred.
Derangements of the lining in any of the pathways connecting the arm or eye to the larger body include the carotid arteries, peripheral arteries, cerebrospinal fluid ducts, etc. In the case of carotid or cephalo-cephalo-arteries, pain may be associated with flushing to the face or back of the head, dry eye, or blurred vision.
In lesions caused by intracellular inflammation, the sites involved may be vascular pathways, such as the carotid or posterior carotid arteries, and/or if the lesions are posterior it may include the rectal arteries.
In lesions caused by proliferative inflammation, the sites involved may be central veins, small veins, renal arteries, microvascular areas, penile arteries, etc. Constriction of these structures may affect renal output, hypotension, stroke, hypertension and hypertension-related stroke.
The plaques may not only affect the blood vessel walls but may also affect the cartilage that lies in contact with them. In DPU, ulcers or clots may develop along with tissue infection that may cause disease progression. In cPP lesions, ulcers may develop along with inflammation of the liver that can lead to cirrhosis.
The presence of inflammation can be differentiated by elevated amounts of the biomarkers albumin-A (the serum antigen A1) and albumin-A18 (albumin-A18 in the blood). When these high levels occur, it may indicate a malignant process in the location of the vessel wall abnormality.